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Morgellons disease: How to Manage It

Morgellons disease: How to Manage It

Morgellons is mysterious and arguable. Here you will find answers to common questions on Morgellons— and suggestions for dealing with it.

Morgellons is quite uncommon and unexplained disease characterised by sores, creeping sensations on and below the skin, and filaments rising from the sores. It isn’t quite  sure what these strings really are. Some say they’re wisps of cotton thread, most likely deriving from clothing or bandages. Others suggest that they result from an infectious process within the skin cells. more study is surely required.

Signs and symptoms

People who have Morgellons usually report the subsequent signs and/or symptoms:

  • Skin rashes or skin sores that could cause itchiness
  • Sensations of crawling on and below the skin
  • Fibers or black stringy material in and on the skin
  • Fatigue
  • Difficulty in concentrating
  • Short-term memory loss

The intense itchiness and open sores related to Morgellons could interfere with an individual’s quality of life.

How widespread is Morgellons disease?

Morgellons disease is a rare condition that usually affects middle-aged white women. A cluster of various cases occurred in California, that prompted the authorities to carry out an investigation study evaluate if those cases were somehow linked to each other. A different study conducted in the UK, went through 5 years of cases, from years 2003 to 2008, and found eighteen patients with a diagnosis of inexplicable dermopathy or Morgellons. Eighty three percent were middle aged women and sixty nine percent were white.

How to Cope with Morgellons

The signs and symptoms related to Morgellons are often distressing. Despite the fact that health professionals might disagree regarding the character of the condition, you deserve an effective. Here is how you can manage your signs and symptoms:

Look for a a doctor that can acknowledge your concerns, that carries out a detailed examination and that can talk through the treatment choices with you.

You must be patient. Your doctor would most likely look for known conditions that lead to evidence-based treatments before considering a diagnosis of Morgellons malady.

Be open minded. Discuss your doctor’s recommendations for treatment — which could also include mental health therapy.

Seek treatment for any other conditions. You could get treatment for other condition that could affect your thinking or your behavior.

Of course if you have any questions about Morgellons you can contact us through the live chat button on the bottom right, or via our contact us form.

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Residents experience swimmers’ itch from Lake Champlain

Residents experience swimmers’ itch from Lake Champlain

Original Source: http://www.shelburnenews.com/2015/09/17/residents-experience-swimmers-itch-from-lake-champlain/

A swim in Lake Champlain on a hot, late-summer day can cause many things: happiness, a sense of well-being, and for some people, an itchy rash. Over the last few weeks, at least a dozen swimmers in the Charlotte and Shelburne area, many at the Charlotte Beach, have experienced an unfortunate side-effect of enjoying the town’s lovely beach.

Swimmer’s itch, or cercarial dermatitis, is an allergic reaction to a parasite that thrives in certain conditions and can be found in lake water. Other than an ick factor, there is no danger or health concern from this parasite or rash. When the water is warm, a parasite can thrive in and attempt to burrow into people’s skin. Though the parasite cannot survive in humans, their burrowing can cause an allergic reaction, which manifests as a rash.

Neil Kamman, Program Manager at the Vermont Department of Environmental Conservation Watershed Management Division, said, “Swimmer’s itch is actually the result of a naturally occurring parasitic organism that in its normal life cycle alternately inhabits snails, or the legs of swimming ducks. Thus, if you have snails (which are naturally occurring), and ducks (which are also naturally occurring), you may have the presence of swimmer’s itch.”

Kamman said that there is no office that regulates or tracks instances of this parasite, but that in extreme circumstances, control of one or the other of the host organisms could be warranted. He also said that in Lake Champlain, not much could be done to stop the parasite’s life cycle, but that perhaps not feeding the ducks and making the area less appealing to them might help.

The Charlotte Beach is a prime breeding ground for the parasite. Eric Howe, an environmental analyst and the Lake Champlain Basin Program Technical Coordinator, said people swimming in shallow, warm water that’s recently been visited by a large flock of ducks or geese, or is near a marshy area that also has a lot of snails, are most susceptible to intercepting the parasite as it passes through the snails and moves out into the water column.

Though there is no way to completely prevent the parasites from attempting to burrow into your skin, he recommends thoroughly toweling off immediately after getting out of the water, which removes the parasites from your skin. Both Howe and Kamman note that the parasite is not only common in Lake Champlain, but is found all over the world.

Though the rash is not contagious, and poses no long-term health issues, it is an itchy, uncomfortable condition. People afflicted can manage their symptoms with anti-itch cortisone cream, Benadryl, oatmeal baths, and other typical remedies for an uncomfortable rash. For more information about the parasites, or cercarial dermatitis, visit http://www.cdc.gov/parasites/swimmersitch/faqs.html.

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Parasitic pox: Swimmer’s itch; where it lurks, how to prevent it

Parasitic pox: Swimmer’s itch; where it lurks, how to prevent it

Source: https://www.stgeorgeutah.com/news/archive/2015/06/23/kss-swimmers-itch-is-active-preventive-measures-treatments/#.VY1wtFUxFGE

SOUTHERN UTAH – If you’re like many people, there is nothing more inviting on a hot summer day than taking a drive out to the lake and dipping into the glistening water … except for one irritating parasite in some waters that thrives and writhes when the shallows get warm, resulting in swimmer’s itch, an irritating and sometimes painful skin rash caused by microscopic parasites.

Swimmer’s itch is not life-threatening and there are preventive measures you can take allowing you to enjoy the water.

“Not everyone gets the swimmers itch but my poor son did ..,” Sonja Ceja wrote in a comment thread on St. George News Facebook page June 13, 2014 | Photo courtesy of Sonja Ceja, St. George News
“Not everyone gets the swimmers itch but my poor son did ..,” Sonja Ceja wrote in a comment thread on St. George News Facebook page June 13, 2014 | Photo courtesy of Sonja Ceja, St. George News

What is Swimmer’s Itch?

The Centers for Disease Control describes swimmer’s itch, or “cercaria,” as a skin rash that is caused by an allergic reaction to microscopic parasites that are carried by waterfowl, semi-aquatic mammals and snails.

As a part of their life cycle, these parasites are released by infected snails into the water. This is where they come in contact with people and burrow into their skin, causing an allergic reaction and rash.

Swimmer’s itch is found throughout the world and is more frequent during summer months.

The good news is, because these larvae cannot develop inside a human, they soon die. Your body’s immune system detects the organism as a foreign protein, then attacks and kills it shortly after it penetrates your skin. The itching and welts are not caused by the organism living under your skin, but by an allergic reaction.

While some people may show no symptoms of swimmer’s itch, others swimming at the same time and place may break out severely. And, much like poison ivy, your sensitivity to swimmer’s itch will increase with each exposure.

Swimmer’s itch cannot be spread from person-to-person, and a swimmer is highly unlikely to get swimmer’s itch from a swimming pool as long as the pool is well maintained and chlorinated.

Where is Swimmer’s Itch active?

Swimmer’s Itch is currently active at Sand Hollow Reservoir.

Water at Sand Hollow reached 80 degrees Monday, making it prime environment for the free-swimming microscopic parasite to flourish; that, and the surrounding alkaline soil, Department of Natural Resources Park Manager Laura Melling said.

The park asks people experiencing swimmer’s itch to report it to park staff at the entrance station. Over the last three weeks, Melling said, she had two cases reported, then five to seven cases, and then two more just since Sunday. But, she said, it’s early. And these don’t account for those who develop the itch after they leave the park or don’t report it.

The parasite lives in shallow water, but the more boats and watercraft are stirring up the lake, the more the parasites are carried throughout the lake. It’s not uncommon for there to be 500 boats on the lake some days, Melling said.

“We run over 22,000 boats through the park a year,” Melling said, “I have a morning crowd, a noon crowd, an afternoon and evening crowd; they come for a couple hours then they leave.”

Neighboring Quail Creek Reservoir does not experience many swimmer’s itch complaints because the water is slightly more acidic which naturally repels the parasite. There have been one or two reported cases of swimmer’s itch from Quail, Melling said, but only when people were up at the top where the springs come into the lake and all the trees grow.

A reliable indication of the parasite is whether or not cattails can be found growing around the water.  Where there are cattails, Melling said, there are swimmer’s itch parasites.

Preventive measures

There are things you can do to reduce your odds of getting swimmer’s itch.

Stapley Pharmacy, in downtown St. George, 102 E. City Center Street, and in the Dino Crossing mall at 446 S. Mall Drive, carries a swimmer’s itch cream used as a preventive measure. The cream is a zinc oxide-type cream that serves as a protective barrier, Pharmacist Brett Petersen said.

“It’s a preventative,” Petersen said. “You apply it before you go in the water – to any skin that will be in the water for more than five minutes.  … If you’re going to be out in the water, you need to reapply after about 90 minutes for it to be effective.”

The Swimmer’s Itch cream is also a sunscreen. An eight-ounce jar costs $17.99.

Other measures:

  • Do not swim in areas where swimmer’s itch is a known problem or where signs have been posted warning of unsafe water
  • Do not swim near or wade in marshy areas where snails are commonly found
  • Do not attract birds to areas where people are swimming by feeding them
  • Apply sunscreen lotion before going in the water — not the spray on kind which is too thin to deter the parasite
  • Towel dry or shower immediately after leaving the water
  • When you get out of the lake, don’t let the water evaporate off your skin. The organism in the droplets of water on your skin will look for somewhere to go as the droplet of water evaporates

Symptoms of the itch

Symptoms of swimmer’s itch may include: tingling, burning or itching of the skin, small reddish pimples or small blisters.

“Within minutes to days after swimming in contaminated water, you may experience tingling, burning, or itching of the skin,” according to the CDC website. “Small reddish pimples appear within twelve hours. Pimples may develop into small blisters.”

Even though itching may last up to a week or more, and will gradually go away, it’s important to remember not to scratch the itch. Scratching the infected areas may result in secondary bacterial infections.

Treating the itch

There are several over the counter remedies your pharmacist can recommend to help relieve the discomfort, but see your physician for a definitive diagnosis.

Most cases of swimmer’s itch do not require medical attention, according to the CDC. If you have a rash, you may try the following for relief:

  • Use corticosteroid cream
  • Apply cool compresses to the affected areas
  • Bathe in Epsom salts or baking soda
  • Soak in colloidal oatmeal baths
  • Apply baking soda paste to the rash – made by stirring water into baking soda until it reaches a paste-like consistency
  • Use an anti-itch lotion
  • Besides anti-itch creams or lotions like hydrocortisone, Petersen recommended taking Benadryl, an over-the-counter antihistimine.

“If we can prevent it,” Petersen said, “that’s the best.”

St. George News Editor-in-Chief Joyce Kuzmanic and reporter Hollie Reina contributed to this report.

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Itchy Skin Rash | Mysterious Skin Problem Causes Itching, Loose Fibers

Imagine your skin burning and itching, and feeling like bugs are crawling under or on top of it.

Imagine having open sores on your face and body. Then imagine having stringlike fibers literally coming out of your skin.

That’s exactly what patients with a mysterious illness called Morgellons disease say happens to them.

Anne Dill is just one of thousands of patients across the country who suffer from these strange symptoms.

Sometimes it feels as if there’s something moving under her scalp, she said, and fibers come out of her skin.

“There’s this fibrous material,” Dill said. “It’s in layers. It’s — I feel like it isolates itself. I think there’s pockets of it.”

Dill said she was reluctant to talk about the illness because she knew that some people would think she was crazy.

“Oh, I know, because right away that’s what I know that they’re gonna say. ‘Uh, there’s no such thing,’” Dill said.

That’s exactly what most doctors do say: As far as they know, Morgellons is not a recognized disease, at this point, at least.

“I’ve seen colors of some of these fibers. Some of them are bright blue,” said Dr. Vincent De Leo, program director of the dermatology department at St. Luke’s-Roosevelt Hospital Center in New York.

“There is nothing in the body that is bright blue. So it has to be something from the environment. And some of them are fibers, but they’re fibers I believe from the environment, not from inside the skin.”

What about the open sores?

De Leo and many others believe the lesions are self-inflicted, caused by scratching because the patients have a psychiatric disorder where they wrongly believe their bodies are infested with parasites.

Morgellons Disease Doctor

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Morgellons disease is mysterious and controversial. Here you’ll find answers to common questions about Morgellons disease — and suggestions for coping with it.

By Mayo Clinic staff

Morgellons disease is a mysterious skin disorder characterized by disfiguring sores and crawling sensations on and under the skin. Although Morgellons disease isn’t widely recognized as a medical diagnosis, experts from the Centers for Disease Control and Prevention (CDC) are investigating reports of the condition, which they refer to as unexplained dermopathy.

If you suspect that you have Morgellons disease, you may have many questions about the condition. Here’s what you need to know about Morgellons disease, including practical tips for managing your signs and symptoms.

What are the signs and symptoms of Morgellons disease?

People who have Morgellons disease report the following signs and symptoms:

  • Skin rashes or sores that can cause intense itching
  • Crawling sensations on and under the skin, often compared to insects moving, stinging or biting
  • Fibers, threads or black stringy material in and on the skin
  • Severe fatigue
  • Inability to concentrate and short-term memory loss
  • Behavioral changes
  • Joint pain
  • Vision changes

Morgellons disease shares characteristics with various recognized conditions, including Lyme disease, liver or kidney disease, schizophrenia, drug or alcohol abuse, and a mental illness involving false beliefs about infestation by parasites (delusional parasitosis).

How widespread is Morgellons disease?

Reports of Morgellons disease have been made in every state in the United States and 15 countries around the world. Most reported cases are clustered in California, Texas and Florida.

What do researchers know about Morgellons disease?

Beyond anecdotal reports, researchers know little about Morgellons disease. The CDC reports no known causes of Morgellons disease and no successful treatment for the condition. Whether Morgellons disease is contagious remains a mystery.

How controversial is Morgellons disease?

Current attitudes toward Morgellons disease fall into various categories:

  • Some health professionals believe that Morgellons disease is a specific condition that needs to be confirmed by future research.
  • Some health professionals believe that signs and symptoms of Morgellons disease are caused by another condition, often mental illness.
  • Other health professionals don’t acknowledge Morgellons disease or are reserving judgment until more is known about the condition.

Some people who suspect Morgellons disease claim they’ve been ignored, criticized as delusional or dismissed as fakers. In contrast, some doctors say that people who report signs and symptoms of Morgellons disease typically resist other explanations for their condition.

How can you cope with the signs and symptoms of Morgellons disease?

The signs and symptoms linked to Morgellons disease can be distressing. Even though health professionals disagree about the nature of the condition, you deserve compassionate treatment. While research continues, take positive steps to manage your signs and symptoms.

  • Establish a caring health care team. Find a doctor who acknowledges your concerns and does a thorough examination. Since Morgellons disease often requires frequent follow-up visits, a local health care team may be most convenient.
  • Be patient. Your doctor will likely look for known conditions that point to evidence-based treatments before considering a diagnosis of Morgellons disease.
  • Keep an open mind. Consider various causes for your signs and symptoms, and follow your doctor’s recommendations for treatment — which may include long-term mental health therapy.
  • Seek treatment for other conditions. Get treatment for anxiety, depression or any other condition that affects your thinking, moods or behavior.
  • Keep track of the latest news about Morgellons disease.Supplement the information you find online with articles published in peer-reviewed medical journals. Remember that some sources are more reputable than are others.

To learn more about Morgellons disease or to report suspected cases of Morgellons disease, call the CDC Morgellons information and voice mail line at 404-718-1199.

The Morgellons Cure

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NCS a Toxicity Disorder with Creeping and Crawling Symptoms

NCS a Toxicity Disorder with Creeping and Crawling Symptoms

On the Diagnosis and Management of Neurocutaneous Syndrome (NCS), A toxicity disorder from dental sealants Omar M. Amin, B.Sc., M.Sc., Ph.D.*

Abstract:

Neurocutaneous syndrome (NCS), a newly discovered toxicity disorder, is characterized by neurological sensations, pain, depleted energy and memory loss as well as itchy cutaneous lesions which may invite various opportunistic infections. Components in the calcium hydroxide dental sealants Dycal, Life and Sealapex have been identified as sources of the observed symptoms. Sulfonamide and neurological toxicity issues are discussed and three case histories are presented. Additional notes on zinc oxide, Fynal, IRM and Sultan U/P sealers are also included. Diagnostic and management protocols at the Parasitology Center, Inc. (PCI) are proposed.

Introduction

The original description of the neurocutaneous syndrome (NCS)1 was “introductory in nature.”1 Examination of many NCS patients and a careful study of their symptoms, exposures, clinical conditions and histories made it possible to identify the underlying cause of the syndrome and proceed with its management.

Materials and Methods

Patients were personally evaluated and their clinical history, records, symptomology and exposures carefully examined. Specimens provided or collected at the Parasitology Center, Inc. (PCI) were studied. An NCS status was only determined based on symptoms and determination that one or more of the suspect sealers have been used on prior dates. Sensitivity to sulfa and elevated levels of sulfa in the blood were used as a confirmation of sulfonamide toxicity. Continuing patients follow our recommendations for dental rehabilitation, extraction of suspect liner(s), and replacement with ethyltoluene sulfonomide (ETS) and zinc oxide free sealants. A list of vitamin/mineral supplements for patient use during the transitional period and another list of substitute sealants are provided.  Patients are followed up to monitor and insure the resolution of  symptoms.

Results and Discussion

The Neurocutaneous Syndrome

The disorder is double faceted with dermatological and neurological symptoms compatible with classical sulfa toxicity. The latter is characterized by changes in blood values, photosensitive reactions, allergic vasculitis sores, bacterial flora changes, and redness of the skin, which may lead to liver and kidney failure.2 The neurological aspects are characterized by pin-prick and/or creeping, painful and irritating movement sensations, often interpreted as parasite movements in various body tissues and/or cavities.. Movement sensations are either unipolar or bipolar and may proceed horizontally or vertically. They may manifest as variably shaped bruises or waves of elevated ripples or channels. In no case was the movement sensation related to parasites1. Neurological symptoms may also include loss of memory, brain fog, lack of concentration and control of voluntary movements.

Fig. 1.  Early NCS sores on the thigh of KM. She was born in 1964, treated with Dycal in two teeth in 1982 and in one tooth in 2002. Neurological symptoms in upper quadrant started in 1997. Cutaneous symptoms began in Spring 2002 preceded by extensive treatment with topical sulfa preparations for possible “mite infestation.” Dycal was removed in December, 2002 and recovery is in progress.

The cutaneous aspects include small itchy sores (Fig.1), inflamed often elevated pimples (Figs.2,3), and fully inflamed and painful open/amorphous mucoid lesions that often enlarge and coalese (Fig.4). Histopathological sections of lesions (Fig.5) show superficial and deep perivascular infiltrate of lymphocytes, accompanied by interstitial deposits of granular mucin material. Eosonophils are usually present within the inflammatory infiltrate and foci of epidermolytic hyporkeratosis are often identified within the epidermis (Fig.5). Lesions may also be on the scalp where they may be associated with infestation of springtails (Collembola). 1 In many cases, lesions are associated with edematous reaction usually in the arms and legs (Fig.6). Blood vessels may also become enlarged and elevated, and head may become hot and turn red. The gum tissue and the teeth and oral mucoid secretions may turn gray and become compromised first and stay compromised the longest. The above creeping sensation is clearly distinguished from these caused by nematodes such as Toxocara canis3 or Dioctophyme sp.4

General symptoms usually include fatigue, compromised immune system, psychological trauma and loss of self- esteem. The depressed immune status in most patients appears to pre-empt them for opportunistic infections.

Compounding Factors

Fig. 2.  Elevated sores on the forehead of KM (Fig.1); note the hot red color of the skin.

 

 

 

Fig. 3.  Diffuse NCS sores covering the whole body that was treated with Dycal in 1985 (Case no. 1)

While NCS itself is not a contagious condition, superimposed opportunistic infections on open sores may be. Initial infection with fungus or bacteria appear to attract subsequent infestations with many arthropod species, especially springtails (Collembola: Insecta).1,5,6,7 Black specks associated with such infections appear to be metabolic waste (fecal elements) of these organisms or mycelial masses of certain fungal species. Staphylococcus aureus, S. haemolyticus,  Streptomyces spp., Candida albicans and Madurella spp. among others, have been identified from cultured swabs taken from sores of various NCS patients. These opportunistic infections have been shown to aggravate the cutaneous symptoms of NCS patients. The Madurella infections are usually associated with black grains of mycelial masses that may be related to the black specks and fibers observed by some NCS patients. The healing of certain patient’s lesions9 was observed to be proportional to the exit of remaining fibers from lesions.3 Patients experiencing complete remission remain susceptible to fungal promoting conditions in damp, shaded, moldy places.

Arthropods identified from sores include fleas, caterpillars, wasps, ants, beetles, winged flies, midges, thrips, ticks, mites, spiders, and springtails.1,4 Springtails may have close association with sores in many NCS patients but they, and other opportunistic infections, are not causal factors of NCS sores.

The Sealants

The three major calcium hydroxide sealants causing NCS (Dycal, Life and Sealapex) considered 9 include only about 50% calcium hydroxide in the catalyst (Table1). Of the components common to all three sealants, ethyltoluene sulfonamide as well as zinc oxide are considered most toxic. Toluene is a known potent nerve toxin.10 The sulfonamide component of this compound causes a sensitivity allergic- toxic reaction ultimately manifesting as the vascular mucoid sores characteristic of the NCS, especially in sulfa sensitive patients.

Fig. 4.  Mucoid NCS/lesions on the face of MM.  She was born in 1950, poisoned with Fynal in six teeth in 1981 and in one tooth in 1986 as well as with Life in two teeth in 1985 and 1988.   Fig. 5.  Histopathological section of one of the roughly 300 sores covering the body of SK. She was born in 1956 and reacted with typical NCS symptoms to a  zinc oxide cement (combined with Durelon) underneath a total veneer job in 1982. The section shows hyperkeratosis –like perivascular dermatitis with eosinophils.           Fig. 6.  Cutaneous sores and swelling in the right hand and arm of DB. Born in 1965, DB had 10 amalgam restorations in 1982 and 1983 using Life. She started experiencing symptoms including ulcerated rash all over the body, unilateral edema and pin-prick and subcutaneous movement sensations in 2001-2002. Life is being removed and recovery is in progress.

Zinc oxide was shown to be genotoxic11, cytotoxic12,13, killing microphages14, and causing chronic

and fibrous inflammatory reaction15,16 ulcerations16 and osteosclerosis.17 Additionally, the toxic effects of zinc oxide and calcium hydroxide were shown to be similar.18,19 Calcium hydroxide was shown to cause periapical inflammation, typical granuloma and partial lack of healing.20 Titanium dioxide and Barium ions (Table 1) were also shown to provoke strong foreign body and bio-incompatible reactions in live tissue.21,22

Cytotoxicity of Dycal, Life and Sealapex was clearly demonstrated invivo and invitro in various tissues.23 Sealapex was shown to cause severe inflammatory infiltration15,24,25 and edema25 accompanied by subcutaneous tissue necrosis15,26 and progressive differentiation and reaction of monocytes, macrophages and epithelial cells27. The final phase of the inflammation is characterized by an intense granulomatus reaction especially in epithelial cells causing various intensities of irritation.28The cytotoxicity29,30 and neurotoxicity31 of Sealapex was well demonstrated in various mammalian systems.

As with Sealapex, Dycal was also shown to cause hemorrhage and acute to consistent inflammatory cells16,32,33 necrosis,16,32,33 tissue loss,33 karyorrhexis,16 neurotoxicity.34 and formation of serous exudates.16 Life has been the least researched sealant. It, however, has the same toxic ingredients, i.e., ethyltoleune sulfonamide and zinc oxide, as Sealapex and Dycal and has been associated with classical NCS symptoms in some of our patients, e.g., DB (Fig.6) and MM (Fig.4).

Sealants not containing ethyltoluene sulfonamide but including zinc oxide and eugenol have also been associated with NCS cases.These include Fynal(>75% zinc oxide), IRM and Sultan U/P (<50% zinc oxide). Fynal was associated with the cases of MM (Fig.4).  Similarly, IRM (by Dentsply caulk) and Sultan U/P (by Sultan Chemists) were associated with classical NCS symptoms in some of our patients.

Table 1.  Components in Catalysts (C) and bases (B) of Dycal. Life and Sealapex

Case Histories

Case #1.

A white female born in 1951. In 1985 she underwent dental repairs, which included the use of Dycal in 20 teeth. The lady is allergic to sulfonomides, with IGE values reaching 5000. Every dental treatment was followed by aggressive skin reactions of allergic and toxicological nature (Fig.3). All tests for parasites were negative. Her symptoms fulminated into full blown typical sulfa toxicity reactions including oozing skin and nasal sores with bloody scabs and smelly discharge and an infection with S. aureus ( Fig.7). Other symptoms included loss of memory, kidney pain and urgency, sensitivity to light and electricity fields, pin-prick and moving sensations under the skin, and swelling. After each treatment, the white female felt totally knocked out with breathing and talking difficulties. She subsequently developed intestinal problems and her skin sores flared up with unbearable and unresolved itching. Photosensitive reactions presented as blotchy skin ( Fig.7) with severe burning sensations in the face, throat and chest.

Dycal was removed in 1991-1992 and initially replaced with Harvard cement. The lady was confined to bed with whole body musculo-skeletal system pain, bowel disturbances and signs of polyneuropathy. Shortly after the removal of the Dycal in February 1992, most of her sores and rashes disappeared and she could tolerate sunlight (Fig.8).

Fig. 7.  Case no. 1 before treatment; note the hot red face.         Fig. 8.  after recovery.

 

 

 

 

Case # 2.

Born in Chicago in 1965, JM was a healthy active Caucasian woman until she started experiencing her first symptoms in 1991. By then, she already had 17 fillings. No sealants were used in one filling; Dycal was used in the other 16. Her earliest symptoms appeared as skin break outs on the face and neck, which was recurrent over the following 9 years, accompanied by body tremors, sleeplessness and joint pain with occasional vomiting of black bile. Thrush appeared in the mouth and around the lips. Pain at the teeth roots persisted throughout the nineties associated with rapid major decay. A sensation of prickling pain with a pressure and movement under the skin, urticaria and skin ulcerations would last for weeks or months. JM’s body showed random swelling with red marks in serpentine-like shapes. The swellings eventually bottlenecked at the knees and ankles. The chest burned and hurt with strange fits of coughing. JM then started losing hair as she experienced night fevers and sweats, and peeling of the skin.

During the early 1990’s JM was medicated with various antibiotics, antiparasitics and herbal remedies. She experienced some anti-inflammatory relief and occasional temporary clearing of ulcers after which ulcers returned and lasted longer. In 1998, massive ulcers appeared on JM’s face at the nasiolobial area and on the skin ( Fig.9). A CBC in 1999 was unremarkable except for a high level of Alpha 1- Globulin of 0.5 (Normal range 0.2-0.4) and low levels of IgA of 99 (normal range 60-400) and IgG of 724 (normal range 700-1500). The right ocular cavity was severely painful and JM was beginning to lose her eyesight.

A major dental repair was completed in 2001 when Dycal was removed from all 16 teeth. Initially, JM experienced a few episodes of sickness, sweats, and vomiting. After the fourth visit, her eyebrow area had a dramatic reduction in swelling, sensation of movement and in the red-hot congestion of her face. JM’s teeth were subsequently rebuilt with gold onlays section by section. By the end of the total repair, Nov.2001, JM has regained her normal skin (Fig.10) with no movement sensations or pain anywhere in her body. This state of total resolution has lasted to date without regression or relapses.

Fig. 9.  Case no. 2 (JM) before treatment; note the lesion

on the right cheek and the hot red face. Fig. 10.  JM after recovery.

 

 

 

 

 

Case #3.

LG, a medium- built white American born in 1957, was in perfect health until September 18, 1998 when she had a filling in her tooth no. 18 using Dycal as a liner. She experienced severe headache within 2 hours. By 6:00 pm she was vomiting and delirious with the headache persisting. Her blood pressure then was monitored at 169/108 and remained high for the following three years despite repeated attempts to control it with Atenenol and Diazide. LG never experienced high blood pressure or headaches before. An MRI scan was negative. In 1999 LG’s health deteriorated progressively with arthritis- like symptoms in her back, heart palpitations, mitral valve prolapse, fatigue, abnormal pap-smears including pre-cancerous cell abnormalities, night sweats, missed periods, and severe depression.  By March 2001, LG, who normally weighed 120 lbs has lost 20 lbs.

In April 2001 lesions started appearing on LG’s face, which quickly became red-hot.  Her legs became swollen and painfully burning. By May 2001, LG had several open lesions (6 mm to 2 cm in diameter) with some surrounding erythema, on her face and scalp. Her cheek pulsated as the facial lesions seemed to track to the chin (Fig.11) where the most fulminating lesion was; nearest to her teeth. The face was burning hot. Springtails (Collembola) and fibers were recovered from these sites. At that time, she showed low lymphocytes of 15.0% (normal 20-43%), high granulocytes of 77.1% (normal 51-74%) and high rheumatoid factor of 22.6 (normal <20 IU/ml). She also tested negative for all communicable diseases then. Her weight dropped to 92 lbs as she started experiencing movement sensations under the skin of her arms, face and scalp. Grayish pustular secretions oozed and moved down from the bloody lesions on the scalp and face. The lesion then extended to her legs.

In January 2002, LG was diagnosed with NCS by OMA. She was allergic to sulfa and sulfonamide compounds. Following our protocol, LG had the filling and the Dycal liner removed from tooth #18 in April 2002. These were replaced with Starflow and Aria (a combination of Bisgma, Tegdma, Lidma and catalysts). Our recommended vitamin supplementation program was initiated then. By May 2002, all symptoms were resolved (Fig.12). Constitutional and neurological functions as well as psychological, emotional and energy levels were restored to normalcy.

Fig. 11.  Case no. 3 (LG) before treatment.         Fig. 12.  LG after recovery; note the return of the natural

“baby” skin back after healing of all facial lesions.

Conclusion

The toxicity of Dycal, Life and Sealapex has been well demonstrated in invivo and invitro studies of various animal and human models by many workers. The toxicity assumed cytotoxic, genotoxic, neurotoxic, phototoxic, necrotic, and inflammatory manifestations compatible with the pathology and symptoms observed in NCS patients. Ethyltoluene sulfonamide, common to all three sealants, is considered the primary cause of the NCS. The toluene component, a known nerve toxin, is believed to be responsible, at least in part, for the neurological symptoms. Neurological abnormalities are related to nerve damage associated with vasomotoric reactions due to a direct influence on the peripheral nerve endings.35 The sulfonamide component is the cause of the cutaneous symptoms, especially in sulfa-sensitive patients who usually had elevated sulfonamide/sulfa levels in blood tests and allergy to sulfa in skin sensitivity tests. The relationship between sulfonamide and phototoxicity has been well established.29 Resolving the symptoms (effect) by removing the sealants (cause) in patients undergoing treatments, confirms this cause-effect relationship.

The nature of causation of NCS precludes contagious transmission. Any similarities of symptoms among partners within the same household are traceable to the transmission of opportunistic infections, especially fungi.

It is recommended not to rehabilitate more than two or three teeth per month. The patient is given a list of vitamins and other supplements to take during the procedure and for the following few weeks until symptoms are completely resolved. After reaching the state of normalcy, the patient may still retain some sensitivity to moldy places lacking sun and fresh air circulation.

After additional test results become available and a satisfactory diagnosis of an NCS case is made at the Parasitology Center, Inc. (PCI), arrangements for dental rehabilitation are made and patient prognosis is monitored.

Acknowlegment

I am grateful to Marie Erixon, Nordea, Sweden for her contributions to the better understanding of issues related to NCS.

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Norrsells N. Aven svenska tandlakare tillats nu sedan EU-intradet att anvanda den effektiva N2-metoden for rotfyllig. Med denna metod kan 500 miljoner kr sparas arligen at patientena och lidandet minskas. Endod Sverige 2002; 5p.

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See also: Amin, O. M. 2004. Dental Sealant Toxicity:  Neurocutaneous Syndrome (NCS), a dermatological and neurological disorder.  Holistic Dental Association Journal (No. 1, Jan.):  1-15  http://www.holisticdental.org/.

See also: Amin, O. M. 2004. On the diagnosis and management of neurocutaneous syndrome, a toxicity disorder from dental sealants. California Dental Association Journal 32 (9): 657-663.

 

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